Montignac

 

Montignac är tydligare med vad som är orsaken till fetma än Atkins och LCHF-företrädare.

Det är hyperinsulinism som är orsaken till fetma och övervikt. Vi har för mycket insulin i kroppen. Bukspottkörteln är utarbetad.

Atkins trodde från början att alla kolhydrater höjde blodsockret lika mycket. Men Montignac uppmärksammade att så inte är fallet. Näringsämnena höjer blodsockret olika mycket. De har högre eller lägre GI.

Det räcker att äta en kost som är låg i GI för att få bukt med hyperisulinismen.

Det är skillnaden mellan Atkins och Montignac.

Hyperinsulinism is what makes us gain weight

As remarked by numerous authors, one of the essential properties of insulin is that it acts on fatty metabolism. This is known as lipogenesis.

• Storage of fatty acids in fat reserve 
  Insuline, and a fortiori hyperinsulinism, stimulates enzyme activity, namely that of lipoprotein lipase. This enzyme’s function is to movilize circulating fatty acids (which correspond to fats eaten in the most recent meal)  in order to stock them as triglycerides, thus increasing the volume of fat cells (adipocytes).
• Additionally, insulin will cause inhibition in another enzyme, triglyceride lipase,. This enzyme is responsible for lipolysis, namely, freeing stored fat reserves. (see below.)

• Storing glucose in fat reserves

If the glycemia peak following a meal is way too high, the corresponding glucose will most probably exceed our body’s needs. The hyperinsulinism provoked by said hyperglycemia will, under the impulse of lipoprotein lipase, convert this residual glucose into fat which will be stored in fat cells.

Hyperinsulinism is definitely the functional cause of weight gain!

http://www.montignac.com/en/la_methode_scientifique.php

 

 För att råda bot på detta räcker det att hålla sig till kolhydrater med ett GI på högst 35.

Weight-losing process (LIPOLYSIS)

As we can see, lipogenesis is the metabolic process which results in fat reserves, and so, in gaining weight. Lipolysis, is exactly the opposite: it is the metabolic process which results in freeing fats, and so, in losing weight.

Our organism finds itself in a situation that forces it to seek fat in fat cells les cellules graisseuses (adypocites) to use as carburant, thus reducing their volume. In order for this to work, insulin has to be low. The mechanism is as follows: low levels of insulin activate the triglyceride-lipase enzyme, which is responsible for evicting fatty acids from adipose tissues (adypocites) by bringing them into our bloodstream so that they can be used as carburant. Our organism will then try to use them (burn them) by modifying its energetic performance, according to its needs.

As a conclusion, we can say that insulin is what catalyzes weight gaining. Thus, in order to lose weight, we have to control insulin levels by trying to keep them as low as possible. To achieve this, we should try to keep after-meal (post-prandial) glycemia peaks at their lowest.

The only solution will naturally be to eat only low, and preferably very low, GI carbs. Experience indicates that, by eating solely low (35 or lower) GI carbs, insulinic response stays low enough to allow our weight-losing enzyme —triglyceride-lipase— to work and to thus provoke a weight-losing process.